UTorrent WebUI 0.80.1 Free Download [32|64bit] 2022 Download: uTorrent WebUIElectrophysiologic abnormalities associated with ventricular tachycardia (VT) in patients with idiopathic dilated cardiomyopathy (IDCM) include: increased ventricular dispersion of refractoriness, higher ventricular pacing thresholds and less refractoriness than normal in the area of the VT circuit. These observations suggest that IDCM may represent an electrical substrate that is appropriate for reentrant VT. Although studies have shown that hemodynamic impairment is not a necessary criterion for sustained ventricular tachycardia (VT), it has been suggested that the number of wavefronts may be limited by the functional capacity of the myocardium to maintain conduction and thus the number of wavefronts that can be initiated simultaneously. Based on this premise, we developed a finite element model to explore the mechanism of VT in IDCM. The model is based on the finite element method (FEM) that has been successfully used to solve many complex physiological problems such as the wave propagation of a shock wave in a human heart during ventricular fibrillation (VF). In this model, the heart is represented by a three-dimensional, non-uniform, non-linearly elastic and isotropic material. The ionic currents that support conduction are calculated by the finite element method (FEM) using the ANSYS Electromagnetic software package. A uniform electric field is used to analyze the initiation and stability of VT. Computer simulations of this model reveal that the distribution of refractoriness and pacing threshold may be spatially inhomogeneous. Furthermore, the results suggest that the greater the dispersion of refractoriness, the greater the number of unstable wavefronts that can be initiated simultaneously, and, therefore, the greater the number of wavefronts that can be used to destabilize the VT. 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